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Synostose, fetalen Alkoholsyndroms, Arms, Differentialdiagnostik, Therapiemoeglichkeiten
|
DerUnfallchirurg.71000678.ger.abstr_2
|
Sentence: Ueber den seltenen Fall einer proximalen radioulnaren Synostose im Rahmen des fetalen Alkoholsyndroms eines 5 jaehrigen Maedchens wird berichtet . Trotz einer fixierten 30 Grad Pronationsstellung hatte die Patientin eine subjektiv nur minimal eingeschraenkte Funktion des rechten Arms . Wir diskutieren anhand des Fallbeispiels die Schwierigkeit der Differentialdiagnostik und die Therapiemoeglichkeiten fuer diese Art der Erkrankung des Bewegungsapparats .
Instructions: please extract entity words from the input sentence
|
[
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] |
Ueber den seltenen Fall einer proximalen radioulnaren Synostose im Rahmen des fetalen Alkoholsyndroms eines 5 jaehrigen Maedchens wird berichtet . Trotz einer fixierten 30 Grad Pronationsstellung hatte die Patientin eine subjektiv nur minimal eingeschraenkte Funktion des rechten Arms . Wir diskutieren anhand des Fallbeispiels die Schwierigkeit der Differentialdiagnostik und die Therapiemoeglichkeiten fuer diese Art der Erkrankung des Bewegungsapparats .
|
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[
"umlsterm"
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tumor necrosis factor - alpha is a Protein, tissue factor is a Protein, CD36 is a Protein, endothelial selectins is a Protein, TNF - alpha is a Protein, TNF - alpha is a Protein, interleukin-1 beta is a Protein, IL-1 beta is a Protein, beta - actin is a Protein, cytokine genes is a Entity, CAT is a Protein, IL-1 beta is a Protein, promoter / enhancer sequences is a Entity, CAT is a Protein, IL-1 beta is a Protein, transcriptional factor activator protein-1 is a Entity, nuclear factor - kappa B is a Entity, SV40 promoter specific protein-1 is a Protein
|
394_0
|
Sentence: Engagement of the Lewis X antigen (CD15) results in monocyte activation.
We previously reported that monocyte adhesion to tumor necrosis factor-alpha (TNF-alpha)-treated endothelial cells increased expression of tissue factor and CD36 on monocytes. Using immunological cross-linking to mimic receptor engagement by natural ligands, we now show that CD15 (Lewis X), a monocyte counter-receptor for endothelial selectins may participate in this response. We used cytokine production as a readout for monocyte activation and found that CD15 cross-linking induced TNF-alpha release from peripheral blood monocytes and cells from the monocytic cell line MM6. Quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) showed an increase in steady-state TNF-alpha mRNA after 3 to 4 hours of cross-linking. CD15 cross-linking also concomitantly increased interleukin-1 beta (IL-1 beta) mRNA, while no apparent change was observed in the levels of beta-actin mRNA, indicating specificity. To examine transcriptional regulation of cytokine genes by CD15 engagement, a CAT plasmid reporter construct containing IL-1 beta promoter/enhancer sequences was introduced into MM6. Subsequent cross-linking of CD15 increased CAT activity. CD15 engagement by monoclonal antibody also attenuated IL-1 beta transcript degradation, demonstrating that signaling via CD15 also had posttranscriptional effects. Nuclear extracts of anti-CD15 cross-linked cells demonstrated enhanced levels of the transcriptional factor activator protein-1, minimally changed nuclear factor-kappa B, and did not affect SV40 promoter specific protein-1. We conclude that engagement of CD15 on monocytes results in monocyte activation. In addition to its well-recognized adhesive role, CD15 may function as an important signaling molecule capable of initiating proinflammatory events in monocytes that come into contact with activated endothelium.
Instructions: please extract entities and their types from the input sentence, all entity types are in options
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Engagement of the Lewis X antigen (CD15) results in monocyte activation.
We previously reported that monocyte adhesion to tumor necrosis factor-alpha (TNF-alpha)-treated endothelial cells increased expression of tissue factor and CD36 on monocytes. Using immunological cross-linking to mimic receptor engagement by natural ligands, we now show that CD15 (Lewis X), a monocyte counter-receptor for endothelial selectins may participate in this response. We used cytokine production as a readout for monocyte activation and found that CD15 cross-linking induced TNF-alpha release from peripheral blood monocytes and cells from the monocytic cell line MM6. Quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) showed an increase in steady-state TNF-alpha mRNA after 3 to 4 hours of cross-linking. CD15 cross-linking also concomitantly increased interleukin-1 beta (IL-1 beta) mRNA, while no apparent change was observed in the levels of beta-actin mRNA, indicating specificity. To examine transcriptional regulation of cytokine genes by CD15 engagement, a CAT plasmid reporter construct containing IL-1 beta promoter/enhancer sequences was introduced into MM6. Subsequent cross-linking of CD15 increased CAT activity. CD15 engagement by monoclonal antibody also attenuated IL-1 beta transcript degradation, demonstrating that signaling via CD15 also had posttranscriptional effects. Nuclear extracts of anti-CD15 cross-linked cells demonstrated enhanced levels of the transcriptional factor activator protein-1, minimally changed nuclear factor-kappa B, and did not affect SV40 promoter specific protein-1. We conclude that engagement of CD15 on monocytes results in monocyte activation. In addition to its well-recognized adhesive role, CD15 may function as an important signaling molecule capable of initiating proinflammatory events in monocytes that come into contact with activated endothelium.
|
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[
"Entity",
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tumor necrosis factor - alpha is a Protein, tissue factor is a Protein, CD36 is a Protein, endothelial selectins is a Protein, TNF - alpha is a Protein, TNF - alpha is a Protein, interleukin-1 beta is a Protein, IL-1 beta is a Protein, beta - actin is a Protein, cytokine genes is a Entity, CAT is a Protein, IL-1 beta is a Protein, promoter / enhancer sequences is a Entity, CAT is a Protein, IL-1 beta is a Protein, transcriptional factor activator protein-1 is a Entity, nuclear factor - kappa B is a Entity, SV40 promoter specific protein-1 is a Protein
|
394_1
|
Sentence: Engagement of the Lewis X antigen (CD15) results in monocyte activation.
We previously reported that monocyte adhesion to tumor necrosis factor-alpha (TNF-alpha)-treated endothelial cells increased expression of tissue factor and CD36 on monocytes. Using immunological cross-linking to mimic receptor engagement by natural ligands, we now show that CD15 (Lewis X), a monocyte counter-receptor for endothelial selectins may participate in this response. We used cytokine production as a readout for monocyte activation and found that CD15 cross-linking induced TNF-alpha release from peripheral blood monocytes and cells from the monocytic cell line MM6. Quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) showed an increase in steady-state TNF-alpha mRNA after 3 to 4 hours of cross-linking. CD15 cross-linking also concomitantly increased interleukin-1 beta (IL-1 beta) mRNA, while no apparent change was observed in the levels of beta-actin mRNA, indicating specificity. To examine transcriptional regulation of cytokine genes by CD15 engagement, a CAT plasmid reporter construct containing IL-1 beta promoter/enhancer sequences was introduced into MM6. Subsequent cross-linking of CD15 increased CAT activity. CD15 engagement by monoclonal antibody also attenuated IL-1 beta transcript degradation, demonstrating that signaling via CD15 also had posttranscriptional effects. Nuclear extracts of anti-CD15 cross-linked cells demonstrated enhanced levels of the transcriptional factor activator protein-1, minimally changed nuclear factor-kappa B, and did not affect SV40 promoter specific protein-1. We conclude that engagement of CD15 on monocytes results in monocyte activation. In addition to its well-recognized adhesive role, CD15 may function as an important signaling molecule capable of initiating proinflammatory events in monocytes that come into contact with activated endothelium.
Instructions: please typing these entity words according to sentence: tumor necrosis factor - alpha, tissue factor, CD36, endothelial selectins, TNF - alpha, TNF - alpha, interleukin-1 beta, IL-1 beta, beta - actin, cytokine genes, CAT, IL-1 beta, promoter / enhancer sequences, CAT, IL-1 beta, transcriptional factor activator protein-1, nuclear factor - kappa B, SV40 promoter specific protein-1
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Engagement of the Lewis X antigen (CD15) results in monocyte activation.
We previously reported that monocyte adhesion to tumor necrosis factor-alpha (TNF-alpha)-treated endothelial cells increased expression of tissue factor and CD36 on monocytes. Using immunological cross-linking to mimic receptor engagement by natural ligands, we now show that CD15 (Lewis X), a monocyte counter-receptor for endothelial selectins may participate in this response. We used cytokine production as a readout for monocyte activation and found that CD15 cross-linking induced TNF-alpha release from peripheral blood monocytes and cells from the monocytic cell line MM6. Quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) showed an increase in steady-state TNF-alpha mRNA after 3 to 4 hours of cross-linking. CD15 cross-linking also concomitantly increased interleukin-1 beta (IL-1 beta) mRNA, while no apparent change was observed in the levels of beta-actin mRNA, indicating specificity. To examine transcriptional regulation of cytokine genes by CD15 engagement, a CAT plasmid reporter construct containing IL-1 beta promoter/enhancer sequences was introduced into MM6. Subsequent cross-linking of CD15 increased CAT activity. CD15 engagement by monoclonal antibody also attenuated IL-1 beta transcript degradation, demonstrating that signaling via CD15 also had posttranscriptional effects. Nuclear extracts of anti-CD15 cross-linked cells demonstrated enhanced levels of the transcriptional factor activator protein-1, minimally changed nuclear factor-kappa B, and did not affect SV40 promoter specific protein-1. We conclude that engagement of CD15 on monocytes results in monocyte activation. In addition to its well-recognized adhesive role, CD15 may function as an important signaling molecule capable of initiating proinflammatory events in monocytes that come into contact with activated endothelium.
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[
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tumor necrosis factor - alpha, tissue factor, CD36, endothelial selectins, TNF - alpha, TNF - alpha, interleukin-1 beta, IL-1 beta, beta - actin, cytokine genes, CAT, IL-1 beta, promoter / enhancer sequences, CAT, IL-1 beta, transcriptional factor activator protein-1, nuclear factor - kappa B, SV40 promoter specific protein-1
|
394_2
|
Sentence: Engagement of the Lewis X antigen (CD15) results in monocyte activation.
We previously reported that monocyte adhesion to tumor necrosis factor-alpha (TNF-alpha)-treated endothelial cells increased expression of tissue factor and CD36 on monocytes. Using immunological cross-linking to mimic receptor engagement by natural ligands, we now show that CD15 (Lewis X), a monocyte counter-receptor for endothelial selectins may participate in this response. We used cytokine production as a readout for monocyte activation and found that CD15 cross-linking induced TNF-alpha release from peripheral blood monocytes and cells from the monocytic cell line MM6. Quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) showed an increase in steady-state TNF-alpha mRNA after 3 to 4 hours of cross-linking. CD15 cross-linking also concomitantly increased interleukin-1 beta (IL-1 beta) mRNA, while no apparent change was observed in the levels of beta-actin mRNA, indicating specificity. To examine transcriptional regulation of cytokine genes by CD15 engagement, a CAT plasmid reporter construct containing IL-1 beta promoter/enhancer sequences was introduced into MM6. Subsequent cross-linking of CD15 increased CAT activity. CD15 engagement by monoclonal antibody also attenuated IL-1 beta transcript degradation, demonstrating that signaling via CD15 also had posttranscriptional effects. Nuclear extracts of anti-CD15 cross-linked cells demonstrated enhanced levels of the transcriptional factor activator protein-1, minimally changed nuclear factor-kappa B, and did not affect SV40 promoter specific protein-1. We conclude that engagement of CD15 on monocytes results in monocyte activation. In addition to its well-recognized adhesive role, CD15 may function as an important signaling molecule capable of initiating proinflammatory events in monocytes that come into contact with activated endothelium.
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Engagement of the Lewis X antigen (CD15) results in monocyte activation.
We previously reported that monocyte adhesion to tumor necrosis factor-alpha (TNF-alpha)-treated endothelial cells increased expression of tissue factor and CD36 on monocytes. Using immunological cross-linking to mimic receptor engagement by natural ligands, we now show that CD15 (Lewis X), a monocyte counter-receptor for endothelial selectins may participate in this response. We used cytokine production as a readout for monocyte activation and found that CD15 cross-linking induced TNF-alpha release from peripheral blood monocytes and cells from the monocytic cell line MM6. Quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) showed an increase in steady-state TNF-alpha mRNA after 3 to 4 hours of cross-linking. CD15 cross-linking also concomitantly increased interleukin-1 beta (IL-1 beta) mRNA, while no apparent change was observed in the levels of beta-actin mRNA, indicating specificity. To examine transcriptional regulation of cytokine genes by CD15 engagement, a CAT plasmid reporter construct containing IL-1 beta promoter/enhancer sequences was introduced into MM6. Subsequent cross-linking of CD15 increased CAT activity. CD15 engagement by monoclonal antibody also attenuated IL-1 beta transcript degradation, demonstrating that signaling via CD15 also had posttranscriptional effects. Nuclear extracts of anti-CD15 cross-linked cells demonstrated enhanced levels of the transcriptional factor activator protein-1, minimally changed nuclear factor-kappa B, and did not affect SV40 promoter specific protein-1. We conclude that engagement of CD15 on monocytes results in monocyte activation. In addition to its well-recognized adhesive role, CD15 may function as an important signaling molecule capable of initiating proinflammatory events in monocytes that come into contact with activated endothelium.
|
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[
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CXCL8 is a Protein, the levels is a Anaphora, IL-6 is a Protein, IL-6 is a Protein
|
183_0
|
Sentence: Involvement of NF-kappaB in cytokine regulation. Cytokine/chemokine levels were determined using ELISA following incubation of Jurkat T-cells with NAI (1 h) and stimulation (24 h). (A) CXCL8 expression was partially inhibited following PMA stimulation (grey bars), whereas the levels were not altered following stimulation with HK E. coli (black bars), this indicates an induction mainly regulated by AP-1. (B) TNF expression was not affected by NAI. (C) IL-6 release was completely inhibited by NAI following PMA exposure, indicating regulation through NF-kappaB since IL-6 expression was significantly increased in response to HK E. coli than PMA. Statistical significance from the positive control (PMA/HK E. coli) was determined using Student's t-test. (n = 3).
Instructions: please extract entities and their types from the input sentence, all entity types are in options
Options: Protein, Anaphora
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Involvement of NF-kappaB in cytokine regulation. Cytokine/chemokine levels were determined using ELISA following incubation of Jurkat T-cells with NAI (1 h) and stimulation (24 h). (A) CXCL8 expression was partially inhibited following PMA stimulation (grey bars), whereas the levels were not altered following stimulation with HK E. coli (black bars), this indicates an induction mainly regulated by AP-1. (B) TNF expression was not affected by NAI. (C) IL-6 release was completely inhibited by NAI following PMA exposure, indicating regulation through NF-kappaB since IL-6 expression was significantly increased in response to HK E. coli than PMA. Statistical significance from the positive control (PMA/HK E. coli) was determined using Student's t-test. (n = 3).
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[
"Anaphora",
"Protein"
] |
CXCL8 is a Protein, the levels is a Anaphora, IL-6 is a Protein, IL-6 is a Protein
|
183_1
|
Sentence: Involvement of NF-kappaB in cytokine regulation. Cytokine/chemokine levels were determined using ELISA following incubation of Jurkat T-cells with NAI (1 h) and stimulation (24 h). (A) CXCL8 expression was partially inhibited following PMA stimulation (grey bars), whereas the levels were not altered following stimulation with HK E. coli (black bars), this indicates an induction mainly regulated by AP-1. (B) TNF expression was not affected by NAI. (C) IL-6 release was completely inhibited by NAI following PMA exposure, indicating regulation through NF-kappaB since IL-6 expression was significantly increased in response to HK E. coli than PMA. Statistical significance from the positive control (PMA/HK E. coli) was determined using Student's t-test. (n = 3).
Instructions: please typing these entity words according to sentence: CXCL8, the levels, IL-6, IL-6
Options: Protein, Anaphora
|
[
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Involvement of NF-kappaB in cytokine regulation. Cytokine/chemokine levels were determined using ELISA following incubation of Jurkat T-cells with NAI (1 h) and stimulation (24 h). (A) CXCL8 expression was partially inhibited following PMA stimulation (grey bars), whereas the levels were not altered following stimulation with HK E. coli (black bars), this indicates an induction mainly regulated by AP-1. (B) TNF expression was not affected by NAI. (C) IL-6 release was completely inhibited by NAI following PMA exposure, indicating regulation through NF-kappaB since IL-6 expression was significantly increased in response to HK E. coli than PMA. Statistical significance from the positive control (PMA/HK E. coli) was determined using Student's t-test. (n = 3).
|
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[
"Anaphora",
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CXCL8, the levels, IL-6, IL-6
|
183_2
|
Sentence: Involvement of NF-kappaB in cytokine regulation. Cytokine/chemokine levels were determined using ELISA following incubation of Jurkat T-cells with NAI (1 h) and stimulation (24 h). (A) CXCL8 expression was partially inhibited following PMA stimulation (grey bars), whereas the levels were not altered following stimulation with HK E. coli (black bars), this indicates an induction mainly regulated by AP-1. (B) TNF expression was not affected by NAI. (C) IL-6 release was completely inhibited by NAI following PMA exposure, indicating regulation through NF-kappaB since IL-6 expression was significantly increased in response to HK E. coli than PMA. Statistical significance from the positive control (PMA/HK E. coli) was determined using Student's t-test. (n = 3).
Instructions: please extract entity words from the input sentence
|
[
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"O",
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"O",
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] |
Involvement of NF-kappaB in cytokine regulation. Cytokine/chemokine levels were determined using ELISA following incubation of Jurkat T-cells with NAI (1 h) and stimulation (24 h). (A) CXCL8 expression was partially inhibited following PMA stimulation (grey bars), whereas the levels were not altered following stimulation with HK E. coli (black bars), this indicates an induction mainly regulated by AP-1. (B) TNF expression was not affected by NAI. (C) IL-6 release was completely inhibited by NAI following PMA exposure, indicating regulation through NF-kappaB since IL-6 expression was significantly increased in response to HK E. coli than PMA. Statistical significance from the positive control (PMA/HK E. coli) was determined using Student's t-test. (n = 3).
|
[
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[
"Anaphora",
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Maritza is a NOMBRE_SUJETO_ASISTENCIA, Sanchez de Miguel is a NOMBRE_SUJETO_ASISTENCIA, 8374542 is a ID_SUJETO_ASISTENCIA, 89 32670766 01 is a ID_ASEGURAMIENTO, Calle del Aguacate , 44 is a CALLE, Madrid is a TERRITORIO, 28040 is a TERRITORIO, 20/07/1956 is a FECHAS, España is a PAIS, 54 años is a EDAD_SUJETO_ASISTENCIA, 31/05/2011 is a FECHAS, Elias Morán Pascual is a NOMBRE_PERSONAL_SANITARIO, 28 28 91467 is a ID_TITULACION_PERSONAL_SANITARIO, femenino is a SEXO_SUJETO_ASISTENCIA, 54 años is a EDAD_SUJETO_ASISTENCIA, Elias Morán Pascual is a NOMBRE_PERSONAL_SANITARIO, [email protected] is a CORREO_ELECTRONICO
|
353_0
|
Sentence: Datos del paciente.
Nombre: Maritza.
Apellidos: Sanchez de Miguel.
NHC: 8374542.
NASS: 89 32670766 01.
Domicilio: Calle del Aguacate, 44.
Localidad/ Provincia: Madrid.
CP: 28040.
Datos asistenciales.
Fecha de nacimiento: 20/07/1956.
País de nacimiento: España.
Edad: 54 años Sexo: M.
Fecha de Ingreso: 31/05/2011.
Médico: Elias Morán Pascual NºCol: 28 28 91467.
Informe clínico del paciente: paciente de sexo femenino, de 54 años de edad, diagnosticada con edentulismo parcial de ambas arcadas y atrofia alveolar severa clase III de Seibert1 en la región de premaxila edéntula.
El plan del tratamiento consiste en la colocación de 2 perlas de hidrogel esterilizadas en autoclave para la expansión tisular de la premaxila, y un posterior aloinjerto óseo en bloque en el mismo lugar.
Dos semanas después de la colocación de las perlas de hidrogel, se realiza la exposición y retirada de las mismas, con colocación de bloques de aloinjerto marca Biograft®, preformados transquirurgicamente por medio de estereolitografía, se pudo realizar el cierre de la herida sin tensión y sin la necesidad de realizar descargas al colgajo, ni incisiones para liberar tensión del periostio.
Una vez culminado el periodo de oseointegración del bloque (aproximadamente 6 meses), se realiza la colocación de los implantes dentales, planeados inicialmente en una adecuada posición, con muy buen tejido óseo y blando para su rehabilitación.
Responsable clínico: Elias Morán Pascual. email: [email protected]
Instructions: please extract entities and their types from the input sentence, all entity types are in options
Options: ID_TITULACION_PERSONAL_SANITARIO, TERRITORIO, ID_SUJETO_ASISTENCIA, FECHAS, SEXO_SUJETO_ASISTENCIA, CALLE, NOMBRE_SUJETO_ASISTENCIA, PAIS, CORREO_ELECTRONICO, EDAD_SUJETO_ASISTENCIA, NOMBRE_PERSONAL_SANITARIO, ID_ASEGURAMIENTO
|
[
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"O",
"O",
"O",
"O",
"B-CORREO_ELECTRONICO",
"O"
] |
Datos del paciente.
Nombre: Maritza.
Apellidos: Sanchez de Miguel.
NHC: 8374542.
NASS: 89 32670766 01.
Domicilio: Calle del Aguacate, 44.
Localidad/ Provincia: Madrid.
CP: 28040.
Datos asistenciales.
Fecha de nacimiento: 20/07/1956.
País de nacimiento: España.
Edad: 54 años Sexo: M.
Fecha de Ingreso: 31/05/2011.
Médico: Elias Morán Pascual NºCol: 28 28 91467.
Informe clínico del paciente: paciente de sexo femenino, de 54 años de edad, diagnosticada con edentulismo parcial de ambas arcadas y atrofia alveolar severa clase III de Seibert1 en la región de premaxila edéntula.
El plan del tratamiento consiste en la colocación de 2 perlas de hidrogel esterilizadas en autoclave para la expansión tisular de la premaxila, y un posterior aloinjerto óseo en bloque en el mismo lugar.
Dos semanas después de la colocación de las perlas de hidrogel, se realiza la exposición y retirada de las mismas, con colocación de bloques de aloinjerto marca Biograft®, preformados transquirurgicamente por medio de estereolitografía, se pudo realizar el cierre de la herida sin tensión y sin la necesidad de realizar descargas al colgajo, ni incisiones para liberar tensión del periostio.
Una vez culminado el periodo de oseointegración del bloque (aproximadamente 6 meses), se realiza la colocación de los implantes dentales, planeados inicialmente en una adecuada posición, con muy buen tejido óseo y blando para su rehabilitación.
Responsable clínico: Elias Morán Pascual. email: [email protected]
|
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|
353_1
|
Sentence: Datos del paciente.
Nombre: Maritza.
Apellidos: Sanchez de Miguel.
NHC: 8374542.
NASS: 89 32670766 01.
Domicilio: Calle del Aguacate, 44.
Localidad/ Provincia: Madrid.
CP: 28040.
Datos asistenciales.
Fecha de nacimiento: 20/07/1956.
País de nacimiento: España.
Edad: 54 años Sexo: M.
Fecha de Ingreso: 31/05/2011.
Médico: Elias Morán Pascual NºCol: 28 28 91467.
Informe clínico del paciente: paciente de sexo femenino, de 54 años de edad, diagnosticada con edentulismo parcial de ambas arcadas y atrofia alveolar severa clase III de Seibert1 en la región de premaxila edéntula.
El plan del tratamiento consiste en la colocación de 2 perlas de hidrogel esterilizadas en autoclave para la expansión tisular de la premaxila, y un posterior aloinjerto óseo en bloque en el mismo lugar.
Dos semanas después de la colocación de las perlas de hidrogel, se realiza la exposición y retirada de las mismas, con colocación de bloques de aloinjerto marca Biograft®, preformados transquirurgicamente por medio de estereolitografía, se pudo realizar el cierre de la herida sin tensión y sin la necesidad de realizar descargas al colgajo, ni incisiones para liberar tensión del periostio.
Una vez culminado el periodo de oseointegración del bloque (aproximadamente 6 meses), se realiza la colocación de los implantes dentales, planeados inicialmente en una adecuada posición, con muy buen tejido óseo y blando para su rehabilitación.
Responsable clínico: Elias Morán Pascual. email: [email protected]
Instructions: please typing these entity words according to sentence: Maritza, Sanchez de Miguel, 8374542, 89 32670766 01, Calle del Aguacate , 44, Madrid, 28040, 20/07/1956, España, 54 años, 31/05/2011, Elias Morán Pascual, 28 28 91467, femenino, 54 años, Elias Morán Pascual, [email protected]
Options: ID_TITULACION_PERSONAL_SANITARIO, TERRITORIO, ID_SUJETO_ASISTENCIA, FECHAS, SEXO_SUJETO_ASISTENCIA, CALLE, NOMBRE_SUJETO_ASISTENCIA, PAIS, CORREO_ELECTRONICO, EDAD_SUJETO_ASISTENCIA, NOMBRE_PERSONAL_SANITARIO, ID_ASEGURAMIENTO
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Datos del paciente.
Nombre: Maritza.
Apellidos: Sanchez de Miguel.
NHC: 8374542.
NASS: 89 32670766 01.
Domicilio: Calle del Aguacate, 44.
Localidad/ Provincia: Madrid.
CP: 28040.
Datos asistenciales.
Fecha de nacimiento: 20/07/1956.
País de nacimiento: España.
Edad: 54 años Sexo: M.
Fecha de Ingreso: 31/05/2011.
Médico: Elias Morán Pascual NºCol: 28 28 91467.
Informe clínico del paciente: paciente de sexo femenino, de 54 años de edad, diagnosticada con edentulismo parcial de ambas arcadas y atrofia alveolar severa clase III de Seibert1 en la región de premaxila edéntula.
El plan del tratamiento consiste en la colocación de 2 perlas de hidrogel esterilizadas en autoclave para la expansión tisular de la premaxila, y un posterior aloinjerto óseo en bloque en el mismo lugar.
Dos semanas después de la colocación de las perlas de hidrogel, se realiza la exposición y retirada de las mismas, con colocación de bloques de aloinjerto marca Biograft®, preformados transquirurgicamente por medio de estereolitografía, se pudo realizar el cierre de la herida sin tensión y sin la necesidad de realizar descargas al colgajo, ni incisiones para liberar tensión del periostio.
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Responsable clínico: Elias Morán Pascual. email: [email protected]
|
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Maritza, Sanchez de Miguel, 8374542, 89 32670766 01, Calle del Aguacate , 44, Madrid, 28040, 20/07/1956, España, 54 años, 31/05/2011, Elias Morán Pascual, 28 28 91467, femenino, 54 años, Elias Morán Pascual, [email protected]
|
353_2
|
Sentence: Datos del paciente.
Nombre: Maritza.
Apellidos: Sanchez de Miguel.
NHC: 8374542.
NASS: 89 32670766 01.
Domicilio: Calle del Aguacate, 44.
Localidad/ Provincia: Madrid.
CP: 28040.
Datos asistenciales.
Fecha de nacimiento: 20/07/1956.
País de nacimiento: España.
Edad: 54 años Sexo: M.
Fecha de Ingreso: 31/05/2011.
Médico: Elias Morán Pascual NºCol: 28 28 91467.
Informe clínico del paciente: paciente de sexo femenino, de 54 años de edad, diagnosticada con edentulismo parcial de ambas arcadas y atrofia alveolar severa clase III de Seibert1 en la región de premaxila edéntula.
El plan del tratamiento consiste en la colocación de 2 perlas de hidrogel esterilizadas en autoclave para la expansión tisular de la premaxila, y un posterior aloinjerto óseo en bloque en el mismo lugar.
Dos semanas después de la colocación de las perlas de hidrogel, se realiza la exposición y retirada de las mismas, con colocación de bloques de aloinjerto marca Biograft®, preformados transquirurgicamente por medio de estereolitografía, se pudo realizar el cierre de la herida sin tensión y sin la necesidad de realizar descargas al colgajo, ni incisiones para liberar tensión del periostio.
Una vez culminado el periodo de oseointegración del bloque (aproximadamente 6 meses), se realiza la colocación de los implantes dentales, planeados inicialmente en una adecuada posición, con muy buen tejido óseo y blando para su rehabilitación.
Responsable clínico: Elias Morán Pascual. email: [email protected]
Instructions: please extract entity words from the input sentence
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"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"B-NOMBRE_PERSONAL_SANITARIO",
"I-NOMBRE_PERSONAL_SANITARIO",
"I-NOMBRE_PERSONAL_SANITARIO",
"O",
"O",
"O",
"O",
"B-CORREO_ELECTRONICO",
"O"
] |
Datos del paciente.
Nombre: Maritza.
Apellidos: Sanchez de Miguel.
NHC: 8374542.
NASS: 89 32670766 01.
Domicilio: Calle del Aguacate, 44.
Localidad/ Provincia: Madrid.
CP: 28040.
Datos asistenciales.
Fecha de nacimiento: 20/07/1956.
País de nacimiento: España.
Edad: 54 años Sexo: M.
Fecha de Ingreso: 31/05/2011.
Médico: Elias Morán Pascual NºCol: 28 28 91467.
Informe clínico del paciente: paciente de sexo femenino, de 54 años de edad, diagnosticada con edentulismo parcial de ambas arcadas y atrofia alveolar severa clase III de Seibert1 en la región de premaxila edéntula.
El plan del tratamiento consiste en la colocación de 2 perlas de hidrogel esterilizadas en autoclave para la expansión tisular de la premaxila, y un posterior aloinjerto óseo en bloque en el mismo lugar.
Dos semanas después de la colocación de las perlas de hidrogel, se realiza la exposición y retirada de las mismas, con colocación de bloques de aloinjerto marca Biograft®, preformados transquirurgicamente por medio de estereolitografía, se pudo realizar el cierre de la herida sin tensión y sin la necesidad de realizar descargas al colgajo, ni incisiones para liberar tensión del periostio.
Una vez culminado el periodo de oseointegración del bloque (aproximadamente 6 meses), se realiza la colocación de los implantes dentales, planeados inicialmente en una adecuada posición, con muy buen tejido óseo y blando para su rehabilitación.
Responsable clínico: Elias Morán Pascual. email: [email protected]
|
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"\n"
] |
[
"CORREO_ELECTRONICO",
"CALLE",
"NOMBRE_PERSONAL_SANITARIO",
"NOMBRE_SUJETO_ASISTENCIA",
"ID_ASEGURAMIENTO",
"ID_TITULACION_PERSONAL_SANITARIO",
"FECHAS",
"SEXO_SUJETO_ASISTENCIA",
"EDAD_SUJETO_ASISTENCIA",
"ID_SUJETO_ASISTENCIA",
"PAIS",
"TERRITORIO"
] |
au(111 ) is a Chemical, ferrocene is a Chemical
|
27190_0
|
Sentence: Ideal redox behavior of the high-density self-assembled monolayer of a molecular tripod on a au(111) surface with a terminal ferrocene group.
Instructions: please extract entities and their types from the input sentence, all entity types are in options
Options: Chemical
|
[
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"B-Chemical",
"I-Chemical",
"O",
"O",
"O",
"O",
"B-Chemical",
"O",
"O"
] |
Ideal redox behavior of the high-density self-assembled monolayer of a molecular tripod on a au(111) surface with a terminal ferrocene group.
|
[
"Ideal",
"redox",
"behavior",
"of",
"the",
"high",
"-",
"density",
"self",
"-",
"assembled",
"monolayer",
"of",
"a",
"molecular",
"tripod",
"on",
"a",
"au(111",
")",
"surface",
"with",
"a",
"terminal",
"ferrocene",
"group",
"."
] |
[
"Chemical"
] |
au(111 ) is a Chemical, ferrocene is a Chemical
|
27190_1
|
Sentence: Ideal redox behavior of the high-density self-assembled monolayer of a molecular tripod on a au(111) surface with a terminal ferrocene group.
Instructions: please typing these entity words according to sentence: au(111 ), ferrocene
Options: Chemical
|
[
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"B-Chemical",
"I-Chemical",
"O",
"O",
"O",
"O",
"B-Chemical",
"O",
"O"
] |
Ideal redox behavior of the high-density self-assembled monolayer of a molecular tripod on a au(111) surface with a terminal ferrocene group.
|
[
"Ideal",
"redox",
"behavior",
"of",
"the",
"high",
"-",
"density",
"self",
"-",
"assembled",
"monolayer",
"of",
"a",
"molecular",
"tripod",
"on",
"a",
"au(111",
")",
"surface",
"with",
"a",
"terminal",
"ferrocene",
"group",
"."
] |
[
"Chemical"
] |
au(111 ), ferrocene
|
27190_2
|
Sentence: Ideal redox behavior of the high-density self-assembled monolayer of a molecular tripod on a au(111) surface with a terminal ferrocene group.
Instructions: please extract entity words from the input sentence
|
[
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"O",
"B-Chemical",
"I-Chemical",
"O",
"O",
"O",
"O",
"B-Chemical",
"O",
"O"
] |
Ideal redox behavior of the high-density self-assembled monolayer of a molecular tripod on a au(111) surface with a terminal ferrocene group.
|
[
"Ideal",
"redox",
"behavior",
"of",
"the",
"high",
"-",
"density",
"self",
"-",
"assembled",
"monolayer",
"of",
"a",
"molecular",
"tripod",
"on",
"a",
"au(111",
")",
"surface",
"with",
"a",
"terminal",
"ferrocene",
"group",
"."
] |
[
"Chemical"
] |
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